参考文献/References:
［1］VANNUCCINI S, TOSTI C, CARMONA F, et al. Pathogenesis of adenomyosis: an update on molecular mechanisms［J］. Reproductive BioMedicine Online, 2017, 35(5): 592-601.
［2］贾晨阳，汪沙，李金皎，等．细胞黏附因子在子宫腺肌病发病机制中作用的研究进展［J］．现代妇产科进展，2017，26（7）：545-547．
［3］LI Tian, LI Yun-guang, PU De-min. Matrix metalloproteinase-2 and-9 expression correlated with angiogenesis in human adenomyosis［J］. Gynecologic and Obstetric Investigation, 2006, 62(4): 229-235.
［4］CHEN Ning, DU Bao-ying, ZHOU Hao, et al. Abnormal expression of Nrf2 may play an important role in the pathogenesis and development of adenomyosis［J］. PLOS One, 2017, 12(8): e0182773.
［5］郑德璇，段华，汪沙，等．局部粘着斑激酶和钙粘素E在子宫腺肌病子宫内膜中的表达与意义［J］．中国计划生育和妇产科，2018，10 （2）：27–31．
［6］BENAGIANO G, BROSENS I, HABIBA M. Structural and molecular features of the endomyometrium in endometriosis and adenomyosis ［J］. Human Reproduction Update, 2014, 20(3): 386-402.
［7］CARRARELLI P, YEN C F, FUNGHI L, et al. Expression of inflammatory and neurogenic mediators in adenomyosis: a pathogenetic role ［J］. Reproductive Sciences, 2017, 24(3): 369-375.
［8］JIANG C, LIU C, GUO J, et al. The expression of toll-like receptors in eutopic and ectopic endometrium and its implication in the inflammatory pathogenesis of adenomyosis［J］. Scientific Reports, 2017, 7(1): 7365.
［9］GUO Jing, CHEN Li, LUO Ning, et al. LPS/TLR4-mediated stromal cells acquire an invasive phenotype and are implicated in the pathogenesis of adenomyosis［J］. Scientific Reports, 2016, 6: 21416.
［10］LIU Li, CHEN Li, JIANG Cai-xia, et al. Berberine inhibits the LPS-induced proliferation and inflammatory response of stromal cells of adenomyosis tissues mediated by the LPS/TLR4 signaling pathway［J］. Experimental and Therapeutic Medicine, 2017, 14(6): 6125-6130.
［11］BENAGIANO G, BROSENS I. The endometrium in adenomyosis［J］. Women's Health (Hillsdale, N.J.), 2012, 8(3): 301–312.
［12］HERNDON C N, AGHAJANOVA L, BALAYAN S A, et al. Global transcriptome abnormalities of the eutopic endometrium from women with adenomyosis［J］. Reproductive Sciences, 2016, 23(10): 1289-1303.
［13］ZHANG J, YANG J, ROY S K, et al. The cell death regulator GRIM-19 is an inhibitor of signal transducer and activator of transcription 3［J］. Proceedings of the National Academy of Sciences of the United States of America, 2003, 100(16): 9342–9347.
［14］WANG Jing, DENG Xiao-hui, YANG Yang, et al. Expression of GRIM-19 in adenomyosis and its possible role in pathogenesis［J］. Fertility and Sterility, 2016, 105(4): 1093-1101.
［15］CARRARELLI P, YEN C F, ARCURI F, et al. Myostatin, follistatin and activin type II receptors are highly expressed in adenomyosis ［J］. Fertility and Sterility, 2015, 104(3): 744.
［16］GUAN Yong-ge, LIAO Jin-bin, LI Kun-yin, et al. Potential mechanisms of an antiadenomyosis Chinese herbal formula Shaoyao-Gancao decoction in primary cell culture model［J］. Evidence-Based Complementary and Alternative Medicine, 2014(1): 982913.
［17］Takeuchi A, Koga K, Miyashita M, et al. Dienogest reduces proliferation, NGF expression and nerve fiber density in human adenomyosis［J］. European Journal of Obstetrics, Gynecology, and Reproductive Biology, 2016, 207: 157-161.
［18］CHEN Li, LI Cai-xia, GUO Jing, et al. Eutopic/ectopic endometrial apoptosis initiated by bilateral uterine artery occlusion: A new therapeutic mechanism for uterus-sparing surgery in adenomyosis ［J］. PLOS One, 2017, 12(4): e0175511.
［19］NIETO M A, HUANG R Y, JACKSON R A, et al. EMT: 2016［J］. Cell, 2016, 166(1): 21-45.
［20］KHAN K N, KITAJIMA M, HIRAKI K A, et al. Involvement of hepatocyte growth Factor-Induced Epithelial-Mesenchymal transition in human adenomyosis［J］. Biology of Reproduction, 2015, 92(2): 1-11.
［21］AN Min, LI Dong, YUAN Ming, et al. Interaction of macrophages and endometrial cells induces epithelial-mesenchymal transition-like processes in adenomyosis［J］. Biology of Reproduction, 2017, 96(1): 46-57.
［22］OH S J, SHIN J H, KIM T H, et al. β-Catenin activation contributes to the pathogenesis of adenomyosis through epithelial-mesenchymal transition ［J］. The Journal of Pathology, 2013, 231(2): 210-222.
［23］FENG Ting-ting, WEI Shao-bin, WANG Yan, et al. Rhein ameliorates adenomyosis by inhibiting NF-kappa B and beta-Catenin signaling pathway ［J］. Biomedicine & Pharmacotherapy, 2017, 94: 231-237.
［24］LIU Xi-shi, SHEN Min-hong, QI Qiu-ming, et al. Corroborating evidence for platelet-induced epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis ［J］. Human Reproduction, 2016, 31(4): 734-749.
［25］SHEN Minhong, LIU Xishi, ZHANG Hongqi, et al. Transforming growth factor β1 signaling coincides with epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis in mice ［J］. Human Reproduction (Oxford, England), 2016, 31(2): 355-369.
［26］ZHU Bo, CHEN Yu-mei, SHEN Xiao-lu, et al. Anti-platelet therapy holds promises in treating adenomyosis: experimental evidence ［J］. Reproductive Biology and Endocrinology, 2016, 14(1): 66.
［27］周应芳．全面认识子宫腺肌病 ［J］．中华妇产科杂志，2013，48（4）：291-294．
［28］汪沙，段华，周莉，等．子宫内膜-肌层交界区超微结构的改变与子宫腺肌病发病的关系［J］．中华医学杂志，2014，94（27）：2115-2118．
［29］SUN Fu-qing, DUAN Hua, WANG Sha, et al. 17β-Estradiol induces overproliferation in adenomyotic human uterine smooth muscle cells of the junctional zone through hyperactivation of the estrogen Receptor-Enhanced RhoA/ROCK signaling pathway［J］. Reproductive Sciences (Thousand Oaks, Calif.), 2015, 22(11): 1436-1444.
［30］IBRAHIM M G, CHIANTERA V, FRANGINI S, et al. Ultramicro-trauma in the endometrial-myometrial junctional zone and pale cell migration in adenomyosis［J］. Fertility and Sterility, 2015, 104(6): 1475.
［31］WANG S, DUAN H, ZHANG Y, et al. Abnormal activation of RhoA/ROCK-I signaling in junctional Zone smooth muscle cells of patients with adenomyosis［J］. Reproductive Sciences (Thousand Oaks, Calif.), 2016, 23(3): 333-341.
［32］MECHSNER S, GRUM B, GERICKE C A, et al. Possible roles of oxytocin receptor and vasopressin-1 alpha receptor in the pathomechanism of dysperistalsis and dysmenorrhea in patients with adenomyosis uteri ［J］. Fertility and Sterility, 2010, 94(7): 2541-2546.
［33］ZHANG Y, YU P, SUN F, et al. Expression of oxytocin receptors in the uterine junctional zone in women with adenomyosis［J］. Acta Obstetricia et Gynecologica Scandinavica, 2015, 94(4): 412–418.
［34］ZHU Bo, CHEN Yu-mei, ZHANG Hong-ping, et al. Resveratrol reduces myometrial infiltration, uterine hyperactivity, and stress levels and alleviates generalized hyperalgesia in mice with induced adenomyosis［J］. Reproductive Sciences, 2015, 22(11): 1336-1349.
［35］史精华，金力，冷金花，等．子宫腺肌病子宫平滑肌细胞中钾离子通道mRNA的表达及雌、孕激素对其的影响［J］．中华妇产科杂志，2015，50（11）：843-847．
［36］GUO Sun-wei. Fibrogenesis resulting from cyclic bleeding: the Holy Grail of the natural history of ectopic endometrium［J］. Human Reproduction, 2018, 33(3): 353-356.
［37］董千靖，段华，郑德璇，等．子宫腺肌病患者子宫肌层纤维化程度与痛经的相关研究［J］．中华妇产科杂志，2018，53（10）：1–5．
［38］LIU X, DING D, REN Y, et al. Transvaginal elastosonography as an imaging technique for diagnosing adenomyosis［J］. Reproductive Sciences, 2018, 25(4): 498–514.
［39］郎景和．子宫腺肌病的若干问题 ［J］．中国实用妇科与产科杂志，2017，33（2）：129-133．
［40］Lupicka M, Socha B, Szczepańska A, et al. Expression of pluripotency markers in the bovine uterus with adenomyosis［J］. Reproductive biology and endocrinology: RB&E, 2015, 13（1）: 110.
［41］SHILINA M A, DOMNINA A P, KOZHUKHAROVA I V, et al. Characteristic of endometrial mesenchymal stem cells in culture obtained from patient with adenomyosis ［J］. Tsitologiia, 2015, 57(11): 771–779.
［42］LI Jing, DAI Yong-dong, ZHU Hai-yan, et al. Endometriotic mesenchymal stem cells significantly promote fibrogenesis in ovarian endometrioma through the Wnt/beta-catenin pathway by paracrine production of TGF-beta 1 and Wnt1［J］. Human Reproduction, 2016, 31(6): 1224-1235.
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